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A possible association of hypokalaemic periodic paralysis, autoimmune thyroiditis and neuromyotonia

Authors:

MH Arambewela ,

University Medical Unit, LK
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MR Sumanathilaka,

Endocrinology Unit, Teaching Hospital, Karapitiya, LK
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KD Pathirana,

University Medical Unit, LK
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CK Bodinayaka

University Medical Unit, LK
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Abstract

Acute hypokalemic periodic paralysis (HPP), a clinical syndrome characterised by acute systemic weakness and low serum potassium (K+), is a rare but treatable cause of acute limb weakness. Hypokalemia can be caused by K+ loss via the kidneys or extra renal routes mainly the gut, or due to transcellular potassium shifts where extracellular K+ will move into the cell. In the latter situation, although there is hypokalaemia, there is no deficit of K+ in the body. The main causes for intracellular shift of K+ are familial hypokalemic periodic paralysis, thyrotoxic periodic paralysis, barium poisoning, insulin excess and alkalosis [1].

Although the association between thyrotoxicosis and HPP is known, HPP with hypothyroidism is extremely rare. We report a case of hypokalemic periodic paralysis associated with hypothyroidism and neuromyotonia

DOI: http://dx.doi.org/10.4038/cmj.v58i4.6311

Ceylon Medical Journal 2013; 58: 175-176

DOI: https://doi.org/10.4038/cmj.v58i4.6311
How to Cite: Arambewela, M., Sumanathilaka, M., Pathirana, K. and Bodinayaka, C., 2013. A possible association of hypokalaemic periodic paralysis, autoimmune thyroiditis and neuromyotonia. Ceylon Medical Journal, 58(4), pp.175–176. DOI: http://doi.org/10.4038/cmj.v58i4.6311
Published on 28 Dec 2013.
Peer Reviewed

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